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Dr. Jeffrey Tucker | (310) 473-2911 | 11600 Wilshire Blvd. Suite 412 | Los Angeles | CA | 90025
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Ninetynine percent of my new patients still come in to see me because they are in pain. But as I help guide them out of pain they soon realize that what I offer is not simply about pain relief and ’adjusting’ bones. We discuss diet, nutrition, sleep habits, emotions, and exercise. All the therapeutic lifestyle enhancements are part of my practice. But the most rewarding part of my practice is teaching clients corrective exercises to help them bring there body back into a better postural alignment and helping people lose weight.
I believe everything is interconnected – the body, mind, spirit. It happens that my emphasis or specialty is on the body work. I use a lot of fascial release, deep soft tissue therapy, mobilization of joints, the Deep Muscle Stimulator (DMS) and warm laser as my primary modalities. But, nutrition can always be improved and I will talk to each client about this subject and I will make supplement recommendations if I feel that will help your case.
“The first treatment is to teach the patient to avoid what harms him.”
When you come in to see me I will perform an evaluation. My job is to help you manage your painful episode, but to also target & contain the risk factors. The number one risk factor for musculoskeletal injury is a previous injury. That means if you have had one painful low back episode you are more likely to have a second episode. My job is to help you lower that risk. The way I help my clients is by teaching them corrective exercises.
Even very small structural changes, if they occur over time, alter the muscles and joints ability to perform properly. Corrective exercise focuses on fixing the cause of pain, instead of just addressing symptoms, it works where many other remedies fall short.
Weakness and tightness are often attacked with isolated and focused strengthening and stretching protocols that don’t work. The majority of musculoskeletal pain syndromes, both acute and chronic, are the result of cumulative microtrauma from stress induced by repeated movements in a specific direction or from sustained misalignments. We need to teach the body how to develop proper movement so you can get out of pain.
I take CoQ10 daily. Adding CoQ10 in therapeutic doses to some clients regimen seems to help the ‘turning point’. Lots of evidence supports its use for heart and/or circulation problems. CoQ10 has shown promise in the treatment of a variety of neurological diseases including Parkinson’s disease, Multiple Sclerosis, migraine headaches, and others. So what is this magic substance?
CoQ10, also known as Ubiquinone, so named because it is ubiquitous in our cells, is a fat soluble (meaning found in fat, absorbed in fat, and utilized in fat metabolism) “nutrient” that is a catalyst for the production of energy from the mitochondria. Mitochondria produce energy we need to carry out our lives. CoQ10 seems to be the prime catalyst or stimulant to this mitochondrial production of energy and probably its rate-limiting step. Therefore it affects all health processes because one cannot heal, white blood cells cannot migrate to the site of the infection or tumor, and circulation cannot flow properly without adequate energy being generated by our cells. This is the role of CoQ10.
CoQ10 is called an anti-oxidant. If you are on statin drugs, you need to take COQ10…period. Lipitor, Zocor, etc. deplete the body of its CoQ10 stores, probably because being a toxin, it shifts the energy needs of the body in the direction of detoxing the drug, leaving the patient exposed to fatigue, congestive heart failure, and a host of neurological illnesses.
I think at least 200 mg per day in circulatory disorders and cancer and up to 2400 mg per day in neurological illnesses is reasonable.
Coenzyme Q10 affects expression of genes involved in cell signalling, metabolism and transport in human CaCo-2 cells. Groneberg DA, Kindermann B, Althammer M, Klapper M, Vormann J, Littarru GP, Doring F. Biomedical Research Institute, Otto-Heubner-Centre, Charite School of Medicine, Free University and Humboldt-University, D-13353 Berlin, Germany. Int J Biochem Cell Biol. 2005 Jun;37(6):1208-18. Epub 2005 Jan 19.
Abstract: Coenzyme Q10 is an essential cofactor in the electron transport chain and serves as an important antioxidant in both mitochondria and lipid membranes. These findings indicate a prominent role of CoQ10 as a potent gene regulator. The presently identified comprehensive list of genes regulated by CoQ10 may be used for further studies to identify the molecular mechanism of CoQ10 on gene expression. PMID: 15778085
Antioxidant treatment of patients with Friedreich ataxia: four-year follow-up. Hart PE, Lodi R, Rajagopalan B, Bradley JL, Crilley JG, Turner C, Blamire AM, Manners D, Styles P, Schapira AH, Cooper JM. University Department of Clinical Neurosciences, Royal Free and University College Medical School, London, England. Arch Neurol. 2005 Apr;62(4):621-6.
Abstract: This study is intended to evaluate the long-term efficacy of a combined antioxidant and mitochondrial enhancement therapy on the bioenergetics and clinical course of FRDA. Publication Types: Clinical Trial PMID: 15824263
Role of mitochondria in neuronal cell death induced by oxidative stress; neuroprotection by Coenzyme Q10. Somayajulu M, McCarthy S, Hung M, Sikorska M, Borowy-Borowski H, Pandey S. Department of Biochemistry and Chemistry, 277-1 Essex Hall, University of Windsor, 401 Sunset Avenue, Windsor, ON, Canada N9B 3P4. Neurobiol Dis. 2005 Apr;18(3):618-27.
Abstract: Our study suggests that water-soluble Coenzyme Q10 acts by stabilizing the mitochondrial membrane when neuronal cells are subjected to oxidative stress. Therefore, Coenzyme Q10 has the potential to be used as a therapeutic intervention for neurodegenerative diseases. PMID: 15755687
Efficacy of coenzyme Q10 in migraine prophylaxis: a randomized controlled trial. Sandor PS, Di Clemente L, Coppola G, Saenger U, Fumal A, Magis D, Seidel L, Agosti RM, Schoenen J. Headache and Pain Unit, Neurology Department, University Hospital Zurich, Frauenklinikstrasse 26, 8091 Zurich, Switzerland. Neurology. 2005 Feb 22;64(4):713-5
Abstract: We compared CoQ10 (3 x 100 mg/day) and placebo in 42 migraine patients in a double-blind, randomized, placebo-controlled trial. CoQ10 was superior to placebo for attack-frequency, headache-days and days-with-nausea in the third treatment month and well tolerated; CoQ10 is efficacious and well tolerated. PMID: 15728298
Bovine cartilage, coenzyme Q10, and wheat grass therapy for primary peritoneal cancer. Forgionne GA. University of Maryland, Baltimore County, Catonsville, MD 21250, USA. J Altern Complement Med. 2005 Feb;11(1):161-5.
Abstract: The accepted postsurgical first-line therapy for primary peritoneal cancer has been a regime of chemotherapy. This paper reports the case of an 89-year-old female who refused chemotherapy but accepted a nutritional alternative. Results after more than 4 years of the nutritional regime have been encouraging with regards to objective and subjective measures. Publication Types: Case Reports PMID: 15750376
Integrated treatment approach improves cognitive function in demented and clinically depressed patients. Bragin V, Chemodanova M, Dzhafarova N, Bragin I, Czerniawski JL, Aliev G. Stress Relief and Memory Training Center, Brooklyn, New York, USA. Am J Alzheimers Dis Other Demen. 2005 Jan-Feb;20(1):21-6.
Abstract: The purpose of this study was to evaluate the efficacy of an integrative treatment approach on cognitive performance. PMID: 15751450 [Coenzyme Q10: biochemistry, pathophysiology of its deficiency and potential benefit of an increased intake] [Article in French] Malchair P, Van Overmeire L, Boland A, Salmon E, Pierard L, Seutin V. Service de Pharmacologie et CNCM, Universite de Liege. Rev Med Liege. 2005 Jan;60(1):45-51.
Abstract: After a brief reminding of the synthesis and function of coenzyme Q10, this article tries to summarise the current state of knowledge about the consequences of its deficiency and about the potential benefits of an increased intake of this coenzyme. We then describe the arguments in favour of such an increase in cardiac diseases and in Parkinson’s disease. PMID: 15771317
Paraquat induces oxidative stress and neuronal cell death; neuroprotection by water-soluble Coenzyme Q10. McCarthy S, Somayajulu M, Sikorska M, Borowy-Borowski H, Pandey S. Chemistry and Biochemistry, University of Windsor, Windsor, Ontario, Canada. Toxicol Appl Pharmacol. 2004 Nov 15;201(1):21-31.
Abstract: Neuronal cell death induced by oxidative stress is correlated with numerous neurodegenerative diseases, including Alzheimer’s disease (AD), Parkinson’s disease (PD), and stroke. Pretreatment with CoQ10 was able to inhibit ROS generation from isolated mitochondria as well as the collapse of mitochondrial membrane potential. Our results indicate that water-soluble CoQ10 can prevent oxidative stress and neuronal damage induced by paraquat and therefore, can be used for the prevention and therapy of neurodegenerative diseases caused by environmental toxins. PMID: 15519605
Pilot trial of high dosages of coenzyme Q10 in patients with Parkinson’s disease. Shults CW, Flint Beal M, Song D, Fontaine D. Department of Neurosciences, University of California, San Diego, La Jolla 92093-0662, USA. Exp Neurol. 2004 Aug;188(2):491-4.
Abstract: The safety and tolerability of high dosages of coenzyme Q10 were studied in 17 patients with Parkinson’s disease (PD) in an open label study. Publication Types: Clinical Trial; Clinical Trial, Phase II PMID: 15246848 Coenzyme Q10 in patients with end-stage heart failure awaiting cardiac transplantation: a randomized, placebo-controlled study. Berman M, Erman A, Ben-Gal T, Dvir D, Georghiou GP, Stamler A, Vered Y, Vidne BA, Aravot D. Department of Cardiothoracic Surgery, Heart-Lung Transplant Unit, Rabin Medical Center, Beilinson Campus, Potah Tikva, Israel. Clin Cardiol. 2004 May;27(5):295-9.
Abstract: The purpose of the present double-blind, placebo-controlled, randomized study was to assess the effect of CoQ10 on patients with end-stage heart failure and to determine if CoQ10 can improve the pharmacological bridge to heart transplantation. The administration of CoQ10 to heart transplant candidates led to a significant improvement in functional status, clinical symptoms, and quality of life. Publication Types: Clinical Trial, Randomized Controlled Trial PMID: 15188947
I do teach my clients that a temporary increase in symptoms is acceptable, as long as the distal symptoms do not remain worse as a result, or peripheralise.
The most therapeutic exercises (loads) will be those that can be self administered by the client 24 hours a day. These loading strategies may initially result in an INCREASE or NO WORSE response. A temporary increase in symptoms is not a sign to STOP a particular exercise (loading strategy). I suggest clients work at home with what I have taught them to explore the INCREASE or NO WORSE response. I often see clients return with a clear BETTER response a few days later.
For low back clients, I do like to start with attempts at achieving further extension in lying (press ups).
If you have any questions call me on my cell phone.
Personally I train in the morning. KNOW THIS: first thing in the morning we are up to 19mm taller in the seated position compared to the evening. This is due to the fact that during the night the disks between the vertebra in our spines become more hydrated with fluids. You literally are taller in the morning than when you go to bed at night. During the day we lose this liquid from or disks, in fact 90% of it is lost 60-90 minutes after getting out of bed.
Here’s how you can decide – if you perform a flexion movement of the torso (lean forward; touch fingers to the floor) as you get out of bed in the morning you will put up to 300% more pressure/compression on your disks. This can be disastrous for someone suffering from back problems. UNLESS you bend over and maintain a neutral spine.
People suffering from back problems should keep there backs in the “neutral spine” position as much as possible and avoid flexion and flexion with rotation of the torso, especially just after getting out of bed. Wait 60 – 90 minutes if you can’t bend without a neutral spine.
90% of back pain comes from muscle weaknesses and imbalances.
Can a few deep breaths really help calm down your stress level? Absolutely. And scientists have even studied how it works… Deep abdominal breathing stimulates the vagus nerve, which runs from your brain stem down through your abdomen. This is the main nerve of the relaxation response. Once stimulated, the vagus activates a chemical compound called acetylcholine–a neurotransmitter that reduces inflammation and sends messages from your brain throughout your body. And the messages all say, “Relax.” As an added bonus, new research shows that the stimulated vagus nerve also activates stem cells that actually repair brain tissue damaged by inflammation. All of that, just from taking deep breaths.
The ‘big three’ when considering ongoing chronic groin pain are:
It is not uncommon to see two or even all three of these conditions co-existing at the same time. However, there still exists a myriad of other conditions that may be affecting the groin area.
The examination needs to focus on the groin, hip & sacroiliac areas. I’ll teach you the best way to improve strength and inhibition of the adductor muscles.
You’ll need to learn proper stretches and strength training .
One of the main back stabilizers and hip flexors. If you sit all day the psoas becomes rounded like a banana; then, you stand up, the psoas pulls on your back, making you more prone to pain and lower back injury.
Most doctors neglect the psoas because it lies so deep below the surface of the skin, and therefore is hard to locate. Unlike other deep lying muscles, like the piriformis, stretching the psoas and trying to locate the psoas with your fingers is not easy. When you look at where it lies in relation to the pelvis and the role it has to play, you’ll understand that in both hip, groin and lower back injuries quality and feel of the psoas is paramount.
I have many techniques for effective self-treatment and in-office treatment of this troublesome hip flexor. I also use the DMS and laser on the psoas.
Most stand-alone hip problems are straightforward.
The hip joint can also be just one part of a bigger puzzle in a complex lower limb problem, as its functioning has such important consequences both for the low back region and for the knee and ankle. Therefore I check the low back, hips, knees and ankles all together.
The hip joint is usually very mobile, having to withstand both direct loading stresses and large rotational forces with weight-bearing activities. Which makes it especially vulnerable to injury in sports that involve pivoting or twisting movements, such as soccer, football, tennis and golf.
During the examination of the hip I carefully check the range of motion and test the length of the hip muscles.
Two movement pathologies are very common.
If only it were so simple that we could split the problem into one or other type of dysfunction. However in reality it will often be a combination of the two.
I teach corrective exercise techniques designed to help improve hip motion and to resolve muscle imbalances.